Annals of Oncology Advance Access originally published online on November 1, 2005
Annals of Oncology 2006 17(4):720-721; doi:10.1093/annonc/mdj056
| ||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||
© 2005 European Society for Medical Oncology
letter to the editor |
Imatinib for secondary Ph+ acute lymphoblastic leukemia induces response in concomitant GBM
| The first 10% of the full text of this article appears below. |
Imatinib is an inhibitor of tyrosine kinase currently employed for the treatment of chronic myelogenous leukemia and inoperable gastrointestinal stromal tumor (GIST).
Several trials [1
, 2
] have been conducted recently with imatinib as monotherapy for treatment of glioblastoma multiforme (GBM) and preliminar results suggest a good level of efficacy in this
Department of Hematology, Universita' Cattolica del Sacro Cuore, Largo Gemelli 8, 00168 Rome, Italy
* (E-mail: emacat@rm.unicatt.it)