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Annals of Oncology Advance Access originally published online on March 12, 2007
Annals of Oncology 2007 18(4):615-618; doi:10.1093/annonc/mdm065
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© 2007 European Society for Medical Oncology

editorials

The treatment of lymphoma complicating autoimmune disease: two birds with one stone?

TR Mikuls, MD, MSPH

Section of Rheumatology and Immunology, University of Nebraska Medical Center and Omaha Veterans Affairs Medical Center, 983025 Nebraska Medical Center, Omaha, NE 68198-3025, USA

(E-mail: tmikuls@unmc.edu)

The first 150 words of the full text of this article appear below.

Lymphoproliferative diseases, particularly non-Hodgkin's lymphoma (NHL), have increased dramatically in frequency during the last three decades with a more than two-fold increase in NHL incidence from the mid-1970s to the mid-1990s alone. NHL is currently the fifth most common malignancy in the USA with ~60 000 new cases diagnosed annually. Although environmental and genetic risk factors for lymphoma are not well defined, antecedent autoimmune disease has been increasingly recognized as a robust determinant of NHL risk. Rheumatoid arthritis (RA), a systemic autoimmune condition characterized by synovial inflammation and progressive joint deformity, has been associated with a two- to six-fold increase in the risk for NHL (reviewed in [1]). Relative risks for lymphoproliferative disease may be even higher in conditions such as systemic lupus erythematosus (SLE) and Sjogren's syndrome (reviewed in [2]).

Despite much needed insight provided by recent investigations, the precise etiology of NHL in the . . . [Full Text of this Article]


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