Skip Navigation



Annals of Oncology Advance Access published online on July 24, 2009

Annals of Oncology, doi:10.1093/annonc/mdp314
This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow E-letters: Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when E-letters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by Schmidt, M.
Right arrow Articles by Awada, A.
PubMed
Right arrow PubMed Citation
Right arrow Articles by Schmidt, M.
Right arrow Articles by Awada, A.
Social Bookmarking
 Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

© The Author 2009. Published by Oxford University Press on behalf of the European Society for Medical Oncology. All rights reserved. For permissions, please email: journals.permissions@oxfordjournals.org

An open-label, randomized phase II study of adecatumumab, a fully human anti-EpCAM antibody, as monotherapy in patients with metastatic breast cancer

M. Schmidt1, M. E. Scheulen2, C. Dittrich3,4, P. Obrist5, N. Marschner6, L. Dirix7, M. Schmidt8, D. Rüttinger8, M. Schuler2, C. Reinhardt8,* and A. Awada9

1 Department of Obstetrics and Gynecology, Johannes Gutenberg University, Mainz
2 Department of Medicine (Cancer Research), West German Cancer Center, University Hospital Essen, Essen, Germany
3 Third Medical Department, Centre for Oncology and Hematology, CEADDP, Applied Cancer Research, Institution for Translational Research Vienna, Vienna
4 Ludwig Boltzmann-Institute for Applied Cancer Research, Vienna
5 Department of Pathology, St Vinzenz Hospital, Zams, Austria
6 Practice for Oncology and Hematology, Freiburg, Germany
7 Medical Oncology Unit, Oncologisch Centrum Sint-Augustinus, Antwerp, Belgium
8 Clinical Development, Micromet AG, Munich, Germany
9 Institut Jules Bordet, Unité Chemiothérapie, Université Libre de Bruxelles, Brussels, Belgium

* Correspondence to: Dr C. Reinhardt, Staffelseestrasse 2, D-81477 Munich, Germany. Tel: +49-89-895277-301; Fax: +49-89-895277-205; E-mail: carsten.reinhardt{at}micromet.de

Background: High-level expression of epithelial cell adhesion molecule (EpCAM) is associated with unfavorable prognosis in breast cancer. This study was designed to investigate two doses of the fully human IgG1 anti-EpCAM antibody adecatumumab (MT201) in patients with metastatic breast cancer (MBC).

Methods: A total of 109 patients were stratified into high- and low-level EpCAM expression by immunohistochemical staining of primary tumors and subsequently randomly assigned to receive monotherapy with either high- (6 mg/kg every two weeks (q2w)) or low-dose adecatumumab (2 mg/kg/ q2w) until disease progression.

Results: No complete or partial tumor responses could be confirmed by central RECIST assessment. The probability for tumor progression was significantly lower in patients receiving high-dose adecatumumab and expressing high levels of EpCAM (hazard ratio 0.43; P = 0.0057 versus low dose and low EpCAM). Three of 18 patients with highest EpCAM expression treated with adecatumumab developed new metastases up to week 6, compared with 14 of 29 patients with low EpCAM. Most frequent treatment-related adverse events (high dose/low dose) were chills (59%/20%), nausea (55%/18%), fatigue (39%/23%) and diarrhea (43%/7%).

Conclusions: Single-agent adecatumumab shows dose- and target-dependent clinical activity in EpCAM-positive MBC, albeit no objective tumor regression. Further investigation of adecatumumab in patients with EpCAM-overexpressing tumors and lower tumor burden is warranted.

adecatumumab, CD326, EpCAM, human mAb, metastatic breast cancer, MT201

Received for publication February 19, 2009. Revision received May 8, 2009. Accepted for publication May 11, 2009.


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?




Disclaimer: Please note that abstracts for content published before 1996 were created through digital scanning and may therefore not exactly replicate the text of the original print issues. All efforts have been made to ensure accuracy, but the Publisher will not be held responsible for any remaining inaccuracies. If you require any further clarification, please contact our Customer Services Department.